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This disease is accompanied by a depletion of nerve cells in the substantia nigra. It is a neurological condition, in which there is reduction in the dopamine levels of the brain. The outcome is a large number of problems relating to the voluntary movement in muscles. The principal symptom of Parkinson’s disease is impaired physical movement. In addition, some patients develop loss of understanding, and a poor comprehension of involved sentences; which has been ascribed to shortcomings in grammatical processing (Angwin, Chenery and Copland 239 – 240).
It is a long – term degenerative disease. Presently, there is no neuroprotective or regenerative treatment for this disease. The medical management of this disease is consequently, limited to improving the quality of life of individuals afflicted with Parkinson’s disease. Many strategies have been developed to reduce the impact of the disease and to enhance the quality of life of its patients. The recent trend has been to garner the subjective experience of persons suffering from this disease and to develop therapeutic methods that rely on the patient’s perspective (Schrag 151).
Neurosurgery provides considerable relief, in respect of the symptoms of Parkinson’s disease. The chief techniques, employed in such interventions are deep brain stimulation, which involves the exercise of control over specific symptoms, via electronic stimulation. The transmission of anomalous signals is prevented in this technique. The instrument utilized in such stimulation has been termed as a brain pacemaker. Another important technique is thalamotomy, in which some genres of tremor are curbed by effecting a lesion on a part of the brain. Finally, pallidotomy is a surgical procedure that reduces dyskinesias, by means of a cerebral lesion (Parkinsons disease - treatment).
Several drugs are available, which have been seen to be reasonably effective in alleviating the symptoms of Parkinson’s disease.
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The earliest known record of the study of Parkinson’s Disease is mentioned in James Parkinson’s “Essay on the Shaking Palsy”. The article describes the most common features of the disease which remain as the standard by which other types of parkinsonism are compared.
This essay focuses not only on disease itself, but also uses statistics and real life examples, such as Mr. Adms's case to analyze the topic of parkinsons disease. The researcher also analyzes symtoms associated with Parkinson’s disease, such as resting tremors, muscle rigidity and bradykinesia are the three main symptoms and medication Mr. Adams has used.
According to the research findings, it can, therefore, be said that the pathology of Parkinson’s disease is complicated making the treatment and control of the disease a complex task. The complexity of the disease has made it almost impossible for researchers to settle for a single approach to treating and controlling the disease.
It results from loss of brain cells charged with producing dopamine, a chemical responsible for relaying messages between the brain’s substantia nigra and other regions of the brain so as to control human body movement, hence an important chemical in ensuring smooth and coordinated muscle movements.
Also, I did not see cites for the "figures" you mention in the paper so I presume you have these "figures" to add to the paper. I cannot guarantee the document to be plagiarism free because you did not have quotation marks around areas that you took directly from other sources, however, with cites in place, this should not be an issue.
Parkinsons disease (PD) is a common progressive neurodegenerative disorder that is characterized by the loss of striatal dopamine (DA) as a result of degeneration of dopaminergic (DAergic) neurons in sunstantia nigra, thus, produces progressive disability.
The current medications which are used to treat the symptoms of Parkinson’s are Amantadine and Levodopa, which are 2 of several medications to alleviate the symptoms associated with the disease.
The drug known as Amantadine is relatively common as a
Ellis, 210) which causes a huge burden on the sufferer himself, his carers, family and the over all medical systems (Rubenstein, 305)
Its occurrence is seen in less than 50 years of age one out of seven patients, and the occurrence increases with the increase in age. At
Its symptoms include tremors or trembling of the limbs and face while at rest; muscle stiffness/rigidity; slowing of movements or motor actions (Bradykinesia); and impaired coordination or imbalance. Patients