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The impact of this scheme will assist the next generations in designing the best responses to mothers who are victims of sickle-cell anemia (Glassberg, 2011).
The Nursing-family partnership provides local communities with the best home-based approaches specially developed to help a mother from low-income families. The program will be implemented through a comprehensive research initiative to identify mothers with this problem. Medical personnel and Nurses will be deployed in the localities. Each county will have an office where registration will be done. This will be done through professional ethics that require non-disclosure. Visitations will commence after victims have been identified.
The program will provide core services such the Nurse-family partnership will provide several services to victim mothers at the local level. Furthermore, the program offers assistance on family planning so that mothers have few chances of sickle cell babies. In addition to this, the program will give consultancy services where mothers will be expected to seek professional counseling and guidance. Providing free and subsidized drugs to mothers to avoid extreme effects to unborn babies
The Initiative has been conducted in different states over the past four decades. It was fast conducted in California for pregnant mothers with sickle cell anemia. The outcome was successful because the number of sickle cell cases in California reduced by half in a span of three years after the inception of the partnership.
There are consistent program effects of the plans, which include improved prenatal health because drugs were overly effective, fewer infant infections arising from mother-child transmissions, Increased intervals between births due to effective family planning techniques imparted t low-income mothers and improved maternal health due to reduced sickle cell count after further tests were administered. Furthermore, the program offers school readiness for
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Vichinsky reported that acute and painful episodes, which can last from two to seven days, are the most common reason for SCD patients to seek emergent medical care. The believes that this population had an opioid addiction acts as a major barrier preventing patients from receiving effective pain management during acute pain episodes.
Sickle cell affects red blood cells which contain haemoglobin and according to Anon (2009) about 250,000 people are carriers of sickle cell genes. Sickle cell disease also means that if both parents are carriers, then it is more likely for the children to develop either sickle cell disease or to be a carrier of the disease.
However, the carriers will have the potential to pass on the affected gene to their offspring.
The chances of my friend being a carrier are one out of four. Both her grandparents were carrier of mutated CFTR as they were healthy and one of their progeny (uncle) had cystic fibrosis.
This process causes damage to the red blood cell membrane and sometimes the red blood cells get attached in blood vessels. This attachment or adherence of the red blood cells deprives the tissues of oxygen causing ischemia and infarction.The disease is chronic with periodic painful attacks,damage of internal organs with complications of strokes and subsequent shortened lifespan.The average life span of sickle cell patients is just 40 years.
ures like premature destruction of red blood cells, elevated erythropoietin levels and increased erythropoietin levels in the marrow and other sites to compensate for the loss of red cells and accumulation of the products of hemoglobin catabolism due to increased rate of red
Patients affected by SCD need red blood cell transfusions on a regular basis to reduce morbidity and mortality. The transfusions are administered intermittently to control or prevent a serious complication of SCD, and as a perioperative measure. Or, as a chronic
Research shows that one in 12 African Americans and about one in 100 Hispanic Americans convey the sickle cell attribute, which implies they are bearers of the infection. Sickle cell sickness is brought about by a change in the hemoglobin-Beta gene found on chromosome 11.