Aortic Valve Replacement - Essay Example

A., Sauaia, A., Moore, E. E., Haenel, J. B., Burch, J. M., and Lezotte, D. C., 1996) involving multiprofessional medical-nursing work of care (Lederer, J. A., Rodrick, M. L., Mannick, J. A., 1999). As I reflect, I can now arrange the events that were relevant to this patient. On the first admission of the patient from theater to intensive therapy unit, the patient was placed on pressure-control ventilation with 100% oxygen with a PEEP of 10, rate of 12, tidal volume 500, and pressure support of 10.

On estimation at that time, arterial blood gas was initially on pH 7.13, pCO2 of 7.0, pO2 of 21.4 with a base excess of -10. SpO2 was 99.8 and bicarbonate 16.9. Lactate was18. The patients in the intensive therapy unit constitute an extremely heterogeneous population in terms of admission diagnosis, co-morbidities, age, race, sex, and socioeconomic conditions, but one feature is common to almost all of them, cardiopulmonary dysfunction (Kelly, J. L. et al., 1997). During my shift hours, I could easily sense that the environment of ITU.

It provided highly integrated and coordinated care with many novel machines and minute-to-minute therapy and observation. This posting in the ITU could improve my understanding of the physiology of such patients in that, I could observe the changes in the patient's parameters in real time. I could see the changes in central venous pressure with a change in rate of fluid therapy (Stone, P.W, and Gershon, R.R.M., 2006). I observed changes in blood gases when the oxygen concentration and ventilation settings were changed.

I could detect when pharmacotherapeutic interventions would fail to produce intended changes in the cardiac output studies. In short, this. Journal of Trauma; 42: pp. 532-536. Fan, J., Marshall, J. C., Jimenez, M., Shek, P. N., Zagorski, J., and Rotstein, O. D., (1998). Hemorrhagic Shock Primes For Increased Expression Of Cytokine-Induced Neutrophil Chemoattractant In The Lung: Role In Pulmonary Inflammation Following Lipopolysaccharide. Journal of Immunology; 161, pp. 440-447. Gadek, J.E., DeMichele, S.J., Karlstad, M.D., et al., (1999). Effect of enteral feeding with eicosapentaenoic acid, gamma-linolenic acid, and antioxidants in patients with acute respiratory distress syndrome.

Enteral Nutrition in ARDS Study Group. Critical Care Medicine; 27: pp. 1409-1420. Gibbs, C.R., Davies, M.K., and Lip, G.Y.H., (2000). ABC Of Heart Failure: Management: Digoxin And Other Inotropes, Blockers, And Antiarrhythmic And Antithrombotic Treatment. British Medical Journal; 320: 495. Inman, K.J., Sibbald,W.J., Rutledge,F.S., Speechley,M., Martin,C.M., and Clark, B.J., (1993). Does Implementing Pulse Oximetry In A Critical Care Unit Result In Substantial Arterial Blood Gas Savings Chest; 104: 542. Kelly, J. L.

, O'Sullivan, C., O'Riordain, M., O'Riordain, D., Lyons, A., Doherty, J., Mannick, J. A., and Rodrick, M. L., (1997). Is Circulating Endotoxin The Trigger For The Systemic Inflammatory Response Syndrome Seen After Injury Annals of Surgery; 225, pp. 530-541. Lu, Z.