Pathophysiology of Endotoxin Induced Septic Shock - Coursework Example

The essay thesis lies in establishing particular body changes and physical responses or reactions that accrue once the septic shock has been activated in the body. The essay establishes that endotoxins in high populations cause a systemic infection, which in turn triggers a series of body changes accumulating in a septic shock. These changes are of two categories namely, cardiovascular and inflammatory.

Both sets of changes usually cause the collapse or failure of the circulatory system, the state in which the severe septic shock culminates. The essay concludes that the high population of endotoxins only triggers a series of body changes (introducing chaotic circulatory changes) that ultimately overwhelms the cardiovascular system. A septic shock accrues with an overwhelming infection consequent to the infection of gram-negative bacteria. The gram-negative bacteria (GNB) are said to result mainly from the actions of the endotoxins alongside other products produced by the infectious agent.

Once the endotoxins population in the body becomes high enough to demonstrate clear effects in functional body changes, the body is then correctly termed as diseased (Dettenmeier 2003, pp. 518-524). This is where Pathophysiology comes in. Pathophysiology is the scientific study of body changes that accrue after the body has been attacked by a disease-causing agent or after an injury. A septic shock is itself an exhibit of an infection, a body change that occurs after the endotoxins have infected the body (Wang 2010, pp.

298 – 327). Septic shocks usually exhibit themselves with sequestration of the blood inside veins and capillaries (Wang 2010, pp. 298 – 327). When the gram-negative bacteria release a lot of the body endotoxins, it most often results in a septic shock (Wang 2010, pp. 298 – 327). That is when the infection is referred to as an Endotoxin-induced septic shock (EISS) or simply as an endotoxin shock (Wang 2010, pp. 298 – 327).  This essay documents the pathophysiological depictions of a septic shock that has been induced by endotoxins.

To achieve this objective, the essay begins with an examination of what the biological reference ‘endotoxins’ refers to. This section will discuss the source, distribution, and chemical structure of endotoxins in detail. This will help to contextualize how the endotoxins eventually induce a septic shock, which is the next section. After that, the essay then details the Pathophysiology characteristics (the body changes) that are triggered by the septic shock consequent to the endotoxin infection.

The essay will also address some of the variations in the Pathophysiology characteristics of Endotoxin Induced septic shock before a brief conclusion on the Pathophysiology of endotoxin-induced septic shock.