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Macroautophagy in Parkinsons GPR37: Critical Analysis of Scientific Process - Term Paper Example

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This term paper discusses the research that deals with specific pathophysiologic pathways, that are analyzed in the essay and aimed to provide better explanation on the topic of Parkinson’s Disease, wherein overexpressed (GPR37) protein bodies are observed in abundance. …
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Macroautophagy in Parkinsons GPR37: Critical Analysis of Scientific Process
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Download file to see previous pages The term paper deals with specific pathophysiologic pathways to better explain Parkinson’s Disease and demonstrates subjective characteristics, as it does not present the limitation of the study and lacks dual authenticity in portraying all dimensions of the research, even to the extent of refuting the set goals of the study. In detail, it takes into account the probability that accumulation of unfolded protein aggregates is not sole definitive cause for such degenerative status. Through a combination of laboratory techniques and embryonic kidney samples, outcomes of the study reveal that human body’s compensating mechanisms increase significant protein degeneration in motor neural functions. Additionally, several conjecture are presented, such as whether similar protective processes are adequate compensations or other controlled conditions can induce excess GPR37 protein or not. The introduction phase of the paper deals with factual situation on Parkinson’s Disease; giving adequate medical background and moving on to expound on what the paper wants to accomplish. With the hypothesis in place, methodology discussed in the paper seems to have answered some of the salient points in the research, such as the significant role of various treatment reagent in inducing function and clearance of inhibitory proteins (GPR37) in neural networks, as well as confirmation of seemingly direct relation between dysfunctional receptors and presence of autophagic bodies in response to the increase of the former. ...Download file to see next pagesRead More
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