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2014 BC Metabolism Meets 2014 AD Lifestyle - Essay Example

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The essay "2014 BC Metabolism Meets 2014 AD Lifestyle" focuses on the critical analysis of the major consequences for health when the 2014 BC metabolism meets the 2014 AD lifestyle. Environmental change over time has led to evolution and mutation in human metabolic function…
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2014 BC Metabolism Meets 2014 AD Lifestyle
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BC Metabolism Meets AD Lifestyle: What are the Consequences for Health? Supervisor Study Program BC Metabolism Meets AD Lifestyle: What are the Consequences for Health? Environmental change over time has led to evolution and mutation in the human metabolic function. Atherosclerosis, metabolic syndrome and diabetes are evidences of variation in lifestyle and genetic makeup. As opposed to the 2014BC, 2014AD have been depicted with a host of economic distress, innovation, sedentary lifestyle and consumption of industrially manufactured goods. Biotechnological methods has taken over the traditional methods. As a result of change in technology, almost everything has been made available to humans and within reach. In the 2014AD, it is quite possible to spend a good percentage of your lifetime eating junk or canned foods. Because of all the pleasure derived from technology, chemical components of food are bound to yield some consequences. Atherosclerosis Previously, naturally extracted fats were used for cooking as compared to newly manufactured oils .Chemical components of manufactured end products have played a major role in genetic mutation evident in the 2014AD as well as environmental changes (Arnold, 2009). Developing countries has recorded high cases of cardiovascular diseases. Women in comparison to men develop mycordial infection ten years earlier than men do (Critchley & Liu, 2004). Natural products consumed in the 2014BC such as fruits reduced the risk of myocardial infection. Today, nature have been substituted with drugs, chemicals and too many tasty substances, which on the other hand contribute to major genetic mutation among the 2014AD generation (Minamikawa et al, 1998) Accessing Atherosclerosis using Risk Models Apparently, characteristics such as diabetes, hypertension, decreased high-density lipoprotein cholesterol, age and gender was traditionally considered a risk factors for coronary heart disease. Framingham, in 1997, included high-density lipoprotein into his risk estimation model. This in turn led to the concept of clinical risk models where there is a baseline state, a risk factor, and a follow-up state (Boyar, 2006: 96-97). Genes and environment drive both baseline state and follow-up state. Since then, many questions have been raised whether to include variables such as lifestyle, social class and origin in the risk prediction models (Paul, 2007). The variables increase in number with time since there is change in environment, lifestyle and genetic mutation (William, 2009: pg. 18-20). This may imply that someone who lived in the 2014BC was safe from the variables evident in the risk models. This is a characteristic of change from traditional lifestyle to the modern lifestyle. Manifestation of Atherosclerosis in the blood vessels can be shown in the blood vessel below. Lesions are formed when T-cells gets to the intina leading to development of macrophages and smooth cells. The lesions forms foam cells which results to fatty steaks vulnerable to atherosclerosis. A C -Reactive Proteins precipitates C-polysaccharide of Streptococcus pneumoniae. If the C -Reactive Proteins is produced to higher levels, it leads to inflammations such as rheumatoid arthritis and sometimes trauma., Therefore, low levels of C –Reactive secretion will result to minimal infection by atherosclerosis (Wick, 2004: pg. 4-6). Obesity Obesity is a chronic disorder that may occur because of hormonal imbalance or metabolic disintegration. It is characterized with sedentary lifestyle, low socio-economic life and eating habits. Obesity is currently a major problem facing majority of individuals. The 2014AD error has been characterized with genetically modified foods, junk foods and consumption of large amounts of cholesterol. More cholesterol means excess of adipose tissue to a level that increases morbidity and mortality (Devlin, 2007). Healthy living entails consumption of the correct quantity of nutrients and fats. Natural foods such as vegetables has been replaced with industrially manufactured foods. This type of feeding may pose challenge to metabolic procedure of someone who lived in the 2014BC. The 2014AD junk food and manufactured fats contains cholesterol in large quantities, this is the major contributing factor to the rising cases of obesity as compared to the 2014BC biological process and hence the high level of challenge. Possible Obese disorder that can challenge 2014BC metabolism. 1. Hormonal related cancers and large bowels. 2. Sterility or infertility 3. Musculoskeletal problems 4. Type 2 diabetes 5. Psychological illness 6. Respiratory problems 7. Gallbladder disease 8. Cardiovascular disease/stroke/hypertension Potentials of becoming over weight A person who weighs more in comparison to his height has more chance of suffering from diabetes. Over weight may be caused by eating fatty food and lack of exercises or movement. In 2014DC, most people do their tasks online and inside their homes. Other even they perform everything indoors and they usually do not get involved in movements compared to 2014BC when almost everything used to be done manually and therefore they hardly were infected from Diabetes (Janeway & Travers 1999). This is because glucose is utilized in the muscular tissue to perform physical activities and fat in muscular tissue reduces absorption of glucose leading to diabetes. Diet In our daily meals, they should have proper amount of proteins, vitamins and fiber, which are necessary for growth maintenance of the body. In the present world people like consuming fast foods, sweets and fatty foods which increase the risk of the diabetes compared to 2014BC when there were less or no those food staffs (Christopher & Donnel, 2007) . Smoking and alcohol Smoking affects the physical activity of a person and continuous smoking causes diabetes. Diabetes patient who continuous smokes do have early death. Most of this company manufacturing these things has increased in the recent years as compared to 2014BC consequently leading to high prevalence of diabetes in 2014DC diet. Treatment of diabetes can achieved by administration of diabetic pills, weight loss programs and traditional medication, which is basically reversal of diet to the old centuries. Conclusion. Considering the change in diet evidenced today, a person who lived a thousand years down the line is most likely to be challenged by what we consume today. People who lived in the 2014BC will have to handle the challenge of breaking down large volumes of dangerous fats, too much sugar as well as chemicals, which were not present in the food of their time. Their metabolic system will be outdated since they are less adapted to the 2014 feeding habits and hence expose them to too much toxins. The result is serous diabetic problems, obesity and cardiovascular deceases that ultimately leads to death. References Assmann G & Schulte H. 2007. Assessing risk of myocardial infarction and stroke; new data from the Prospective Cardiovascular Muenster (PROCAM) Study. Eur J Clin Invest; 37:925-32. Christopher J. O’Donnell. 2007. (Framingham Heart Study National Heart Lung, and Blood Institute Framingham, MA, and Bethesda, MD, USA William Kannel. 2009.Framingham Heart Study Framingham, MA, US Boston University School of Medicine Boston, MA, USA Page 3-5. Arnold von Eckardstein. 2009. MD Institute of Clinical Chemistry University of Zurich Zurich, Switzerland Page 10-12. Paul Ridker. 2007.MD Brigham and Women’s Hospital Boston, MA, USA Page 15-19. Boyar A. 2006.Creating a web application that combines Framingham risk with electron beam CT coronary calcium score to calculate a new event risk. J Thorac Imaging 21:91-96. Critchley J, Liu J. 2004.Explaining the increase in coronary heart disease mortality in Beijing between 1984 and 1999. Circulation 10:1236-44. Wick, G., Knuflach, M & Xu, Q. 2004.Annual Review of immunology Autoimmune and Inflammatory Mechanisms in Atherosclerosis Chap 22, 361-403. Minamikawa J, Yamauchi M, Inoue D, et al. 1998. Another potential use of troglitazone in noninsulin-dependent diabetes mellitus. J Clin Endocrinol Metab 83:1041–1042. Vasan S. (2006). Biomarkers of cardiovascular disease: molecular basis and practical considerations. Circulation 16; 113(19):2335-62. Devlin Thomas M (2011) Textbook of Biochemistry with Clinical Correlations 7th ed. Hoboken, NJ : John Wiley & Sons, Inc., Library classmark: 572 Janeway, D & Travers, A (1999) Immunobiology: The Immune System in Health and Disease 4th ed. Edinburgh : Churchill Livingstone, Library classmark: 616.079 Read More
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