H. pylori - Research Paper Example

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These infections are dangerous and affect the normal processes that occur in the stomach. However, this bacterium has more benefits to the human body. As seen in the…
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Helicobacter For decades, the government has protected larger endangered species. Should such laws apply to microorganisms such as Helicobacter?
These laws supporting endangered species should be applied to this micro organism. It is well documented that it poses a lot of the benefits to the human race.
How can a bacterium like Helicobacter be both “good” and “bad” for human health?
Helicobacter is unacceptable in the human health in that it infects individuals with a number of infections. These infections are dangerous and affect the normal processes that occur in the stomach. However, this bacterium has more benefits to the human body. As seen in the paper, the bacteria can prevent a lot of diseases such as asthma among children.
How might physicians of the future prescribe Helicobacter as a pro biotic for treating patients?
H pylori absence in the body might lead to an increased risk of numerous diseases. According to various studies conducted, it is known that esophageal diseases presence is high on H pylori negative individuals. Its absence can lead affect the stomachs micro biota. The lack of H pylori mostly among kids has led to increased risk of asthma and allergies (Martin 7).
Virulence factors are structures, chemicals, and metabolic functions that increase the chance an organism will cause disease. C A G (A) and V a C (A) are virulence factors of Helicobacter pylori. Explain how these two proteins mediate infection and disease.
C A G (A) is a marker for strains that increase the chances of one contracting peptic ulcer disease. It symbolizes a human gastric gene. This strain contains a secretion system (type 4) that infuses D N A and proteins in the stomach. C A G (A) product S21 -S23) is in the epithelial cells in vivo and vitro form. Once inside the stream, C A G (A) associates with a tyrosine phosphate (SHP-2) that will affect the spreading of the epithelial cell. This injected C A G (A) interacts with Grb2, and it activates the ERK pathway. This leads to cell scattering and proliferation in the MDCK cells (Martin 6).
V a C (A) causes vesiculation in the cell lines of the epithelial. It posses two signal region, S1 and S2, and two mid regions, M1 and M2. It affects the host by creating pores in the cell membranes leading to urea egress. It also facilitates the loosening of tight epithelial junctions allowing the crossing of nutrients through the mucosal barrier to the pylori gastric niche (Martin 6).
Works Cited
Blaser, Martin J. " Endangered Species in the Stomach." H. Pylori and its Interactions with
Humans (2005): 1-11. Read More
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