Article Analysis Introduction A concussion is a brain injury which is caused by repeated head blows. A concussion can result in loss of consciousness, headaches, long or less hours of sleep, moodiness and loss of recognition. Anyone who experiences more than one concussion can take a longer period to recover (Lazar, 2010).This article discusses a life of an excellent footballer Kevin Turner…
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Assumptions The researchers involved in this case make numerous assumptions concerning the impact of concussions on the development of ALS and CTE symptoms. The assumptions made by researcher might contribute to the misdiagnosis of various patients showing the symptoms. Among the notable assumptions are the following: Concussions lead to development of the symptoms of ALS and CTE. Trauma could lead to development of the ALS condition. The number of concussions might lead to development of the symptoms of ALS. Risk factors leading to ALS condition once individuals suffer multiple concussions. Arguments The arguments of researchers within this research mainly focus on the condition experienced by footballers. Though the researchers refer to conditions experienced by other footballers, Turner’s case appears to have driven the researchers into trying to conduct the research regarding connection between concussions and ALS or CTE conditions. Turner experienced a drastic change from a well organized, intelligent, and loving husband to a shabby and lazy man. Depression and stress coupled with other problems appeared to completely drive Turner’s life towards destruction, and family members thought this was a result of lifestyle changes experienced after retiring from football. McKee, however, notes that these changes in behaviour and personality seem structural, eliminating the possibility of psychological problems. Numerous footballers had been diagnosed with CTE through autopsy, and family members thought the same could be affecting Turner. Appel, however, fails to concur with the deductions made by McKee, claiming that these deductions lack scientific proof to support them. According to Appel, the number of concussions necessary to prove diagnoses of CTE remains scientifically unknown, as well as the risk factors involved in developing of CTE following multiple concussions (McCrory, 2001). Appel completely fails to see the logic behind concluding presence of CTE in an individual who had recovered from concussions several years back. Appel’s argument remains that, since science has not yet proved the relationship between CTE and concussions, the deductions made by McKee require further clarification. Though he does not refuse the possibility of the individual acquiring CTE, he fails to link the condition to the concussions suffered. Appel also seems to suspect that the brains inspected by McKee had both ALS and CTE conditions. He however, wonders how ALS could become connected to concussions, since thirty years of scientific research have failed to prove any possible connections. These researches also fail to provide any link between trauma and ALS, hence magnification of Appel’s doubts on McKee’s observations. These doubts instil fear of having patients being misdiagnosed, as might be the case with McKee’s deductions. McKee also doubts her own conclusions by not being sure whether the presence of tau and TDP-43 proteins within the brain might have caused ASL (McKee, 2010). This conclusion becomes supported by the observation she makes in the individuals who have previously suffered multiple concussions. While some people, having suffered multiple concussions, suffer from ASL others never show the symptoms of ASL. This makes it difficult to ascertain whether concussions really cause ASL. Though people’
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