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Background information Diabetes mellitus 2 is a chronic disease that is characterized by excess sugar levels in blood and is the most reported of the two types of diabetes. Even though it is ideally expected among adults, prevalence has been reported among younger populations because of the effect of obesity. The main cause of diabetes mellitus 2 is the body’s inability to respond to environmental factors towards secretion and consumption of insulin, an element that controls blood sugar level.
This is further associated with the inability of body lipids and the liver to accordingly react to availability of insulin. The disease is majorly associated with age but is also associated with obesity and overweight among the younger population (Medline, 2012). Literature review The main cause of type 2 diabetes is the body’s inability to appropriately produce and use insulin. This is an effect of changed body processes that is caused by inflammation of essential organs and cells that are responsible for the secretion and application of the biochemical insulin.
Contrary to the belief that poor eating habits and lack of exercise is the primary cause of obesity towards type two diabetes, it has been established that some types of obesities, and consequently type two diabetes, are results of non-diet based inflammations. . The author explains that availability of anti-inflammatory agents reduces the realized effects towards a controlled insulin level. As a result, anti-inflammatory elements are recommended for controlling inflammation of the primary cells and organs to ensure efficiency in management of body insulin (Sears, 2009).
Golbini, Badran and Laher also identify the role of inflammation and anti inflammatory factors in controlling and preventing diabetes. They establish this from research findings that individuals with type two diabetes exhibit higher inflammatory conditions as compared to people who are not diabetic. Further, the higher inflammatory level among diabetic patients is a significant health factor because of its association with other complications such as the cardiovascular complications, as it increases incidence of the other chronic diseases.
Exercise is however identified as an anti-inflammatory agent that manages diabetic characteristics in a person. Even though it has a short-term inflammatory effect, exercise works to, in the end, develop anti inflammatory effects that promise a preventive measure against development of diabetes in general and the more prevalent type 2 diabetes in particular. This is because exercise reduces inflammatory factors and facilitates development of anti-inflammatory elements in the body. Myokines, a product of exercise, produced from active muscles, is an example of agents that facilitates controlled inflammations towards regulating type 2 diabetes besides other chronic complications (Golbini, Badran and Laher, 2012).
Exercise also has effects that control C-creative proteins. A regular activity towards controlling the proteins induces anti-inflammatory effects for
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